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primary antibody solution for pdgfrβ 3169 t  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc primary antibody solution for pdgfrβ 3169 t
    Primary Antibody Solution For Pdgfrβ 3169 T, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/primary antibody solution for pdgfrβ 3169 t/product/Cell Signaling Technology Inc
    Average 90 stars, based on 1 article reviews
    primary antibody solution for pdgfrβ 3169 t - by Bioz Stars, 2026-03
    90/100 stars

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    Ses suppresses APAP-induced ferroptosis in mice. (a,b) Flow cytometric analysis and quantification of mitochondrial membrane potential (MMP) in mouse kidney. (c) Iron content was detected in kidney tissues. (d–k) Western blot bands showing the protein expressions and relative signal intensities of solute carrier family 7 member 11 (SLC7A11), glutathione peroxidase 4 (GPX4), transferrin receptor protein 1 (TfR1), ferroportin1 (FPN1), ferritin light chain (FTL), and ferritin heavy chain 1 (FTH1) in kidney tissue. For quantification, the intensity was normalized to β -actin and the control was set to 1. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure group.
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    Characterization of hUC-MSCs. (A) morphology of third-passage hUC-MSCs; (B) tri-lineage differentiation of hUC-MSCs. Oil red O staining after adipogenic differentiation induction of hUC-MSCs, Alizarin red S staining after osteogenic differentiation induction of hUC-MSCs, Alcian blue staining after chondrogenic differentiation induction of hUC-MSCs; (C) flow cytometry analysis of surface markers in hUC-MSCs. The first row (from left to right) represents the expression of CD34, CD45, and HLA-DR. The second row (from left to right) represents the expression of CD73, CD90, and CD105. The last row is the negative control groups for APC and FITC are shown from left to right.

    Journal: Biochemistry and Biophysics Reports

    Article Title: JIB-04 improves PPARγ expression and enhances adipogenic differentiation efficiency in human umbilical cord mesenchymal stem cells

    doi: 10.1016/j.bbrep.2025.102142

    Figure Lengend Snippet: Characterization of hUC-MSCs. (A) morphology of third-passage hUC-MSCs; (B) tri-lineage differentiation of hUC-MSCs. Oil red O staining after adipogenic differentiation induction of hUC-MSCs, Alizarin red S staining after osteogenic differentiation induction of hUC-MSCs, Alcian blue staining after chondrogenic differentiation induction of hUC-MSCs; (C) flow cytometry analysis of surface markers in hUC-MSCs. The first row (from left to right) represents the expression of CD34, CD45, and HLA-DR. The second row (from left to right) represents the expression of CD73, CD90, and CD105. The last row is the negative control groups for APC and FITC are shown from left to right.

    Article Snippet: The cell pellet was resuspended in 95 μL pre-cooled flow cytometry staining buffer, followed by the addition of 5 μL of the prepared primary antibody solution (532222, 531671, Sigma; 17-3714-81, 17-1057-41, 11-4714-82, 11-0459-41, 11-0349-41, ebioscience).

    Techniques: Staining, Flow Cytometry, Expressing, Negative Control

    Ses suppresses APAP-induced ferroptosis in mice. (a,b) Flow cytometric analysis and quantification of mitochondrial membrane potential (MMP) in mouse kidney. (c) Iron content was detected in kidney tissues. (d–k) Western blot bands showing the protein expressions and relative signal intensities of solute carrier family 7 member 11 (SLC7A11), glutathione peroxidase 4 (GPX4), transferrin receptor protein 1 (TfR1), ferroportin1 (FPN1), ferritin light chain (FTL), and ferritin heavy chain 1 (FTH1) in kidney tissue. For quantification, the intensity was normalized to β -actin and the control was set to 1. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure group.

    Journal: Redox Report : Communications in Free Radical Research

    Article Title: Sesamin protects against Acetaminophen-induced nephrotoxicity by suppressing HMOX1-mediated apoptosis and ferroptosis

    doi: 10.1080/13510002.2025.2529695

    Figure Lengend Snippet: Ses suppresses APAP-induced ferroptosis in mice. (a,b) Flow cytometric analysis and quantification of mitochondrial membrane potential (MMP) in mouse kidney. (c) Iron content was detected in kidney tissues. (d–k) Western blot bands showing the protein expressions and relative signal intensities of solute carrier family 7 member 11 (SLC7A11), glutathione peroxidase 4 (GPX4), transferrin receptor protein 1 (TfR1), ferroportin1 (FPN1), ferritin light chain (FTL), and ferritin heavy chain 1 (FTH1) in kidney tissue. For quantification, the intensity was normalized to β -actin and the control was set to 1. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure group.

    Article Snippet: The primary antibodies against β -actin (AC026, 1:50000), solute carrier family 7 member 11 (SLC7A11, A2413, 1:2000), glutathione peroxidase 4 (GPX4, A1933, 1:1500), transferrin receptor protein 1 (TfR1, A5865, 1:1500), ferroportin1 (FPN1, A14884), ferritin light chain (FTL, A11241, 1:1500), ferritin heavy chain 1 (FTH1, A19544, 1:1500) and HMOX1 (A19062, 1:2000) were all sourced from ABclonal (Wuhan, China).

    Techniques: Membrane, Western Blot, Control

    Ses inhibits APAP-induced apoptosis in mice. (a–c) Western blot bands showed the expression of B-cell lymphoma-2 (Bcl2) and Bcl2-associated X (Bax) proteins and the relative signal intensity in kidney tissues. For quantification, the intensity was normalized to β -actin and the control was set to 1. (d) Terminal deoxynucleotidyl transferase (TdT)-mediated deoxyuridine triphosphate (dUTP) nick end labeling (TUNEL) staining of apoptotic cells in kidney tissue sections (scale bar = 50 µm). As indicated by the yellow arrowheads, green fluorescence marks TUNEL-positive signals, blue fluorescence stains the nuclei, and their colocalization in the merged image indicates apoptotic cells. (e) Proportion of apoptosis. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure.

    Journal: Redox Report : Communications in Free Radical Research

    Article Title: Sesamin protects against Acetaminophen-induced nephrotoxicity by suppressing HMOX1-mediated apoptosis and ferroptosis

    doi: 10.1080/13510002.2025.2529695

    Figure Lengend Snippet: Ses inhibits APAP-induced apoptosis in mice. (a–c) Western blot bands showed the expression of B-cell lymphoma-2 (Bcl2) and Bcl2-associated X (Bax) proteins and the relative signal intensity in kidney tissues. For quantification, the intensity was normalized to β -actin and the control was set to 1. (d) Terminal deoxynucleotidyl transferase (TdT)-mediated deoxyuridine triphosphate (dUTP) nick end labeling (TUNEL) staining of apoptotic cells in kidney tissue sections (scale bar = 50 µm). As indicated by the yellow arrowheads, green fluorescence marks TUNEL-positive signals, blue fluorescence stains the nuclei, and their colocalization in the merged image indicates apoptotic cells. (e) Proportion of apoptosis. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure.

    Article Snippet: The primary antibodies against β -actin (AC026, 1:50000), solute carrier family 7 member 11 (SLC7A11, A2413, 1:2000), glutathione peroxidase 4 (GPX4, A1933, 1:1500), transferrin receptor protein 1 (TfR1, A5865, 1:1500), ferroportin1 (FPN1, A14884), ferritin light chain (FTL, A11241, 1:1500), ferritin heavy chain 1 (FTH1, A19544, 1:1500) and HMOX1 (A19062, 1:2000) were all sourced from ABclonal (Wuhan, China).

    Techniques: Western Blot, Expressing, Control, End Labeling, TUNEL Assay, Staining, Fluorescence

    Ses improves APAP-induced renal injury through Heme oxygenase 1 (HMOX1). (a) The docking result analysis of Ses and HMOX1. (b,c) Western blot bands showing the protein expressions and relative signal intensities of HMOX1 in kidney tissues. For quantification, the intensity was normalized to β -actin and the control was set to 1. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure group.

    Journal: Redox Report : Communications in Free Radical Research

    Article Title: Sesamin protects against Acetaminophen-induced nephrotoxicity by suppressing HMOX1-mediated apoptosis and ferroptosis

    doi: 10.1080/13510002.2025.2529695

    Figure Lengend Snippet: Ses improves APAP-induced renal injury through Heme oxygenase 1 (HMOX1). (a) The docking result analysis of Ses and HMOX1. (b,c) Western blot bands showing the protein expressions and relative signal intensities of HMOX1 in kidney tissues. For quantification, the intensity was normalized to β -actin and the control was set to 1. Values are expressed as the mean ± SEM ( n = 6); * p < 0.05 difference from the control group; ** p < 0.01 difference from the control group; *** p < 0.001 difference from the control group; # p < 0.05 difference from the APAP exposure group; ## p < 0.01 difference from the APAP exposure group; and ### p < 0.001 difference from the APAP exposure group.

    Article Snippet: The primary antibodies against β -actin (AC026, 1:50000), solute carrier family 7 member 11 (SLC7A11, A2413, 1:2000), glutathione peroxidase 4 (GPX4, A1933, 1:1500), transferrin receptor protein 1 (TfR1, A5865, 1:1500), ferroportin1 (FPN1, A14884), ferritin light chain (FTL, A11241, 1:1500), ferritin heavy chain 1 (FTH1, A19544, 1:1500) and HMOX1 (A19062, 1:2000) were all sourced from ABclonal (Wuhan, China).

    Techniques: Western Blot, Control